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Miro1 has been implicated in mesenchymal stem cell transfer of mitochondria to damaged lung epithelia following inhibition of the electron transport chain and allows for suppression of the allergic asthma phenotype in mouse models of the disease ( Herein, we developed a novel mouse model to study Miro1 biology in the context of allergic asthma, in which we hypothesize that epithelial deletion of Miro1 leads to exacerbated inflammatory responses following chronic allergen exposure

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Archived from the original on 16 July 2001