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E-Cigarette (2.4% Nicotine) Induces Hepatic Mitochondrial Dysfunction Markers Increased levels of PARP1 activity releases poly (ADP-ribose) polymer from the nucleus to the cytosol, leading to depolarization of the mitochondrial membrane potential (Figure 4A shows an immune blot of PINK1 of hepatic extracts from saline and e-cigarette (2.4% nicotine)-treated mice

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