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The effects of cigarette smoking on cell renewal in the GI tract have been reviewed by a number of studies showing that cigarette smoke and its active ingredients not only inhibit mucosal cell proliferation, but also induce cell apoptosis during ulcer healing (8,24)

To further illustrate the effectiveness of the combined treatment, we employed low-density lipoprotein receptor (VLDLR) knockout mice ( Vldlr / mice), which exhibit characteristics of retinal angiomatous proliferation and choroidal neovascularization

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