Funding The authors gratefully acknowledge the financial supports from the National Natural Sciences Foundation of China (81800386), the Hunan Provincial Natural Science Foundation of China (2021JJ30020), the financial supports from the scientific research project of health commission of Hunan province (202101021784), the financial supports from the science and technology plan project of Hengyang City (202150063459) and College Students Research Learning and Innovative Experiment Plan in University of South China (202110555098, S202110555307, X202110555516, X202110555523, X202110555529, S202210555256, 2022X10555115)
AKT1 participates in ferroptosis vulnerability by driving autophagic degradation of FTH1 in cisplatin-resistant ovarian cancer
GCS, -glutamylcysteine synthetase
The cell-cycle-regulated PLA signals of RSMCSororin phenocopied those of PARP1-RSMC (Fig
In: Freinkel RK, Woodley DT, editors