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Although all isoforms promote excitatory synaptic transmission, their subcellular localization varies significantly, with NLGN3 diffusely distributed in dendrites and NLGN3A1A2 enriched in spines, revealing that splicing-dependent isoform distribution specificity is a key molecular mechanism for bidirectional regulation of inhibitory synapses (85)

However, in stressful, oxidative-compromising conditions, this reductase seems not to be sufficient, underlying the importance of this -glutamyl cycle (37)

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Baillieres Clinical Neurology