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It was found that Schisandrin A suppressed the levels of urea nitrogen and urinary albumin, reduced the release of inflammatory factors, inhibited the level of MDA activity, and increased the levels of SOD, CAT, and GHS in STZ-induced DN, which effectively ameliorated oxidative stress and inflammatory responses in the DN model

53,626 Accumulating evidence demonstrates that aberrant PARPs activation by DNA damage causes NAD + depletion, contributing to the progression of tumorigenesis and neurodegenerative disorders that involve DNA repair defects

Inhibition of HIF-1 reduced blood brain barrier damage by regulating MMP-2 and VEGF during acute cerebral ischemia