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Valproic acid has been characterized as a Histone deacetylase inhibitor, functioning directly transcription of gene levels by blocking the deacetylation of histones and increasing the accessibility of transcription sites

Whereas l-cysteine supplements have shown little to no improvement in GHS levels, NAC has a bioavailability of around 6%

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Impaired magnesium ion binding and carbohydrate kinase activities corresponded with dysfunctional RBKS and PRTFDC1, indicating TCA cycle disruption and mitochondrial oxidative phosphorylation inefficiency [63, 64]

J Trace Elem Med Biol 62:126653