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Presynaptic transient receptor potential channel (TRP channel) receptor activation by PregS modulates glutamate release and increases sEPSC in acutely isolated hilar neurons of the dentate gyrus, an increase that is blocked by TRP channel antagonists (121)

Additionally, this combination produced outcomes comparable to those achieved with 5 mg/kg leflunomide alone, particularly in terms of reduced inflammation (204)

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