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Further studying of the mechanism shows that, in a rat model of doxorubicin-induced nephropathy, HKC can downregulate the protein expression of transforming growth factor (TGF)-1 and the p38mitogen-activated protein kinase (MAPK) by suppressing the p38/ MAPK signaling pathway [37,38]

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The combination of probiotics and oxytocin produced greater improvements in social responsiveness, aberrant behaviors, and clinician-rated global functioning than either treatment alone, and was associated with favorable shift in gut microbiome network structure