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References 1.Ferdinand K.C., Nasser S.A

GSTO1*E155del polymorphism associated with increased risk for late-onset Alzheimers disease: association hypothesis for an uncommon genetic variant

NF-B activation, NLRP3 inflammasome assembly, and production of TNF-, IL-1, nitric oxide, ROS, and complement proteins all contribute to synaptic dysfunction, disrupt mitochondrial dynamics, and increase dopaminergic neuron death

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