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Among those defects, treatment of intrabony defects has been studied and very well researched with randomized controlled clinical trials and long-term studies [16,17,18,19,20,21]

To summarize, an ideal candidate mechanism for counteracting tendency for runaway dynamics imposed by Hebbian-type learning rules on weight changes of excitatory synapses and activity in interneurons should fulfill the following requirements

Mitochondrial dysfunction may therefore be relevant not only to inherited mitochondrial disease, but also to autism, schizophrenia, borderline personality disorder, depression, OCD, cognitive decline, fatigue and treatment nonresponse

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Pediatr Adolesc Endocrinol