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The observation of increased post-prandial GLP-1 concentration and increased risk of T2D in carriers of PAM diabetes risk alleles is counterintuitive with prior studies which demonstrated reduced GLP-1 concentrations in individuals with pre-diabetes and diabetes compared with persons without diabetes [41, 42], although this pattern has been reported in carriers of TCF7L2 diabetes risk alleles who have been termed incretin resistant 43

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