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generated a double-knockout murine model by crossing the G6PD-deficient mice of Pretsch and Charles [62] with the atherosclerosis-prone apo E / mouse [12] and observed reduced aortic superoxide production, lower markers of inflammation, and less atheroma development
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Their research demonstrated that macrophages are central to the pathogenesis of pancreatic diseases, with M1 macrophage polarization being linked to exacerbated inflammation in pancreatitis
Cells treated with test compounds were washed with PBS and incubated with 10 M 2,7-dichlorofluorescein diacetate (DCFH-DA) at 37 C for 30 min, followed by washing with PBS, digestion with trypsin, and collection
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