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In order to treat and prevent ROS in gastric cancer, it may be crucial to focus on the enhancement of ROS by neutralizing antioxidants to induce cancer cell death, and the inhibition of ROS activity or increase of antioxidant capacity to regulate pro-tumorigenic signaling pathways

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However, further investigation is needed to understand how these DEGs and pathways translate into the broader systemic effects that become evident following the engagement of CNS GIPR and GLP-1R

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