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Nevertheless, pharmacological maintenance or restoration of GSH levels can inhibit NF-B signaling and cytokine release, thereby preventing excessive or chronic inflammation.37 Thus, although the present IHC findings suggest possible modulation of NF-Brelated pathways, definitive mechanistic interpretation requires molecular-level confirmation through gene or protein expression analyses

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10.1016/S2213-8587(19)30422-X Summary Keywords GLP-1 receptor agonists, diabetes, coronary artery disease, acute and chronic coronary syndromes, atherosclerosis, coronary microcirculation Citation Pahud de Mortanges A, Sinaci E, Salvador Jr D, Bally L, Muka T, Wilhelm M and Bano A (2022) GLP-1 Receptor Agonists and Coronary Arteries: From Mechanisms to Events