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An apparent paradox is that whereas GLP-1 increases glucose-stimulated insulin secretion in the isolated pancreas fulfilling the definition of an incretin but it lowers insulin secretion in physiologically relevant conditions [6, 10, 18]

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In the CNS, GLP1 is synthesised by a relatively small population of a few thousand neurons primarily located within the lower brainstem of vertebrates, in the nucleus tractus solitarii (NTS) and in the adjacent intermediate reticular nucleus (IRT) (Larsen et al., 1997