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Reduced inflammation, improved biomechanics, and metabolic reprogramming likely act in concert, opening perspectives that go beyond mere mechanical off-loading of the joint

Abstract Antibiotic use disrupts microbial composition and activity in humans, but whether this disruption in turn affects host metabolic health is unclear

Further human studies are required to substantiate the claims

Conclusively, these results suggest that ALC is capable to attenuate kainate-induced SE which is somewhat mediated through its lowering of oxidative stress, neuroinflammation, and pyroptosis that are related to its neuroprotective effect