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Notably, the hepsin / mice exhibited a more pronounced increase in ROS levels after thioacetamide treatment compared with wild-type mice, which aligned with our hypothesis that hepsin / mice experience heightened ROS stress in the early stages of APAP exposure due to their elevated expression of GJ proteins (Fig

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[2] [3] Understanding that food noise stems from biological, hormonal, and psychological originsrather than simply a lack of willpoweris crucial for developing effective, compassionate management strategies that do not rely on pharmacological intervention