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In addition, semaglutide prevented the DM2-induced impairments of pro-opiomelanocortin (POMC), and G-protein-coupled receptor 43 (GPR43) and simultaneously increased the NeuN + and Glucagon-like Peptide-1 receptor (GLP-1R+) neurons in the hippocampus

Originally designed to manage diabetes, semaglutide surprised researchers with its remarkable weight loss effects

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doi: 10.1007/s007260070013