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Recent studies have explored small-molecule inducers, selenium-based compounds, and targeted nanodelivery approaches to restore GPX4 function or mimic its antioxidant capacity in ferroptosis-prone diseases, with therapeutic implications for OA and beyond ( In summary, GPX4 acts not only as a terminal antioxidant enzyme but also as a hub that integrates transcriptional, post-transcriptional, and nutritional cues

5, when PC3 cell lines were treated with SBA-15 MSN and FA-SNCD@SBA-15 at concentrations up to 50 g mL 1 for a duration of 24 h, the cell viability remained above 90%

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doi:10.1016/j.cmet.2019.07.013 337

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