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Proteases rapidly degrade many peptides, exhibit poor oral bioavailability due to enzymatic breakdown in the digestive tract, and suffer from short systemic half-lives, reducing their clinical efficacy

The same group published extensive evidence for BPC-157s tendon-healing effects, including a mechanistic study demonstrating that BPC-157 promotes tendon outgrowth and cell survival through specific signaling pathways involved in angiogenesis and cell migration [2]

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