Astrogliosis is prevalent in the epilepsies, and interestingly a mouse forebrain neuron-specific knockout of SOD2 resulted in seizures, oxidative stress, and marked GFAP and vimentin gene upregulation, indicative of astrogliosis resulting from increased neuronal mtROS ( Discussion Together, these studies examining oxidative stress, redox dysregulation, metabolic alterations, and neuroinflammation suggest that these processes are all not only involved in the pathogenesis of epilepsy, but fundamentally linked (See Scheme 1)
Biochemical and genetic analyses further show that the conserved histone fold domains (HFDs) of both paralogs are sufficient for biological function and complex incorporation
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Additionally, the peptide apparently stimulated dermal keratinocyte proliferation, as evidenced by histological analysis of biopsies
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