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(216) utilized C57BL/6J mice to induce a model of SSc, confirming that mesenchymal stem cells (MSCs) alleviate SSc symptoms by modulating the balance of the gut microbiota

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2g,h), glucagon infusions at any dose failed to significantly stimulate secretion of somatostatin, indicating that glucagon-induced somatostatin secretion depends on both glucagon and GLP-1 receptors, consistent with the finding that glucagon interacts with both receptors [14, 28]