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Mechanistically, liraglutide administration inhibited the upregulation of pain-related markers in the TNC, including calcitonin gene-related peptide (CGRP), c-fos, and components of the PI3K/Akt signaling pathway [94]

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Following short-term exposure to extreme elevations, individuals show increased CD73 activity, which boosts adenosine production, and decreased eENT1 expression, reflecting a rapid and reversible shift in red blood cell response to hypoxia