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Endoplasmic reticulum-mitochondria contacts are prime hotspots of phospholipid peroxidation driving ferroptosis

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Additionally, SPAE modulated the expression of key genes involved in oxidative stress response ( Nrf2 , SOD2 ), apoptosis ( Bax , Bcl-2 ), lipid metabolism ( PPAR- ), and DNA damage ( p53 ), further supporting its protective role at the molecular level