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Moreover, several clinical studies show that glycemia improves when doses are increased but with unacceptable rates of gastrointestinal adverse events 16,17,18 , suggesting that maximum benefits from GLP-1R agonism are in fact not realized with current treatments

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Due to these mutations there are only truncated proteins produced that would be non-functional since they have lost part of the NAD + binding motif and the C-terminal domain that would be responsible for binding L-threonine

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