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By down-regulating PAR1, SalB inhibits coagulation factors, activates the fibrinolytic system, dissolves excessive fibrin deposition, preserves the structural integrity of pulmonary tissues, and suppresses fibrous tissue proliferation (Zhang et al., 2021)

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Gut dysbiosis has been related with the pathogenesis of a wide spectrum of liver diseases including autoimmune hepatitis, autoimmune cholangiopathies, alcoholic liver disease, non-alcoholic liver disease (NAFLD), hepatocellular carcinoma, acute liver injury, and liver fibrosis/cirrhosis (Hartmann et al., 2019)