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To be clear, this doesnt rule out the possibility of any adverse effects caused by the peptide

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Several mechanisms have been reported that explain the regulation of the concentration and activity levels of Keap1, such as transcriptional regulation (STAT6 and hypoxia-inducible factor) (59), miRNAs (miR-7 and miR-24-3p) (54), post-translational regulation, such as ubiquitination (60) and S-nitrosylation (61), as well as degradation of the Keap1 protein (p62-dependent autophagy) (62)