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Differential pathophysiological mechanisms between CHS and EHS While CHS and EHS share core pathogenic pathways (e.g., hyperthermia, inflammation, cell death), their mechanistic features differ significantly due to distinct patient demographics and triggers: Inflammatory kinetics EHS (predominant in young, active individuals) is characterized by rapid, exertion-amplified inflammatory responses

5) via its activity on histone deacetylases and is capable of influencing genomic administration

Superoxide dismutase (SOD) and glutathione peroxidase (GP) activity in erythrocytes are involved in the psoriasis onset (7)

Its molecules are used in creams and pills with the aim of treating overweight and cellulite

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