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Transgenic AD models such as APP/PS1 mice, which overexpress mutant human amyloid precursor protein and presenilin-1, have also revealed mitochondrial fragmentation and respiratory deficits, reinforcing the hypothesis that impaired mitochondrial dynamics and mitophagy are mechanistically linked to AD pathology

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Grx gene silencing in hepatocytes further sensitized hepatocytes to TNF-mediated killing

Peptide therapy takes advantage of the abilities of these short-chain amino acids to act as chemical communicators to our glands so that hormones can be released through a series of subcutaneous injections of the peptide