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They specifically investigated the in vitro induction of eryptosis on healthy RBCs exposed to septic plasma at different time points and evaluated the relationship between eryptosis percentage and Endotoxin Activity Assay (EAA) levels, mortality, and other biological markers of inflammation and oxidative stress [383]

Activation of GPX4 partially rescued these effects, reinforcing the role of mGPDH as an auxiliary mitochondrial defense mechanism against ferroptosis under pathological stress

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Maintaining the balance in bilirubin metabolism and excretion is crucial, as disturbances can lead to pathological conditions, including gallstone formation, due to altered bilirubin handling and metabolism