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These processes play key roles to promote chemoresistance in tumors, where elevated GSH inactivates electrophilic chemotherapeutic agents (cisplatin, alkylators), GSTs enable drug conjugation, and MRP-catalyzed efflux reduces intracellular drug levels - while inhibiting therapy-induced oxidative stress at the same time

A discontinuous drug release profile with a low initial burst and two peaks of drug release at the 2nd and 7th week is observed, corresponding to different mechanisms of exenatide release from microspheres (Figure 5A and B) [79]

Given the variability in compounding practices and dosing accuracy, efficacy cannot be assumed equivalent to SURMOUNT trial data

Hill coefficients approaching unity suggest simple receptor occupancy kinetics without apparent cooperativity