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The release of de novo synthesized proinflammatory mediators CXCL8, CCL2, CCL3, CCL4, and TNF in hiMC was almost completely (90100%) inhibited [74]

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In studies of human glioblastoma, the absence of exogenous Gln significantly reduces glutamate levels, resulting in the conversion of the dimer GLS1 into a self-assembled, extremely low Km filamentous polymer that further depletes intracellular Gln

de Rooij WE, Diks MAP, Warners MJ, Ampting M, van Esch B, Bredenoord AJ