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Mechanistically, vitamin D exerts anti-inflammatory effects via VDR-mediated suppression of TLR signaling, induction of Treg expansion, and downregulation of proinflammatory cytokines such as TNF- and IL-6

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The highly expressed and inducible endogenous NAD (P) H: quinone oxidoreductase 1 in cardiovascular cells acts as a potential superoxide scavenger

Accelerated granulation provides the foundation upon which epithelial cells regenerate