This dual pathway is relevant to cellular aging research because oxidative stress is not a standalone hallmark but a cross-cutting mechanism that accelerates telomere shortening, mitochondrial dysfunction, and the accumulation of senescent cells
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Autophagy 17:20542081 Wenzel SE, Tyurina YY, Zhao J, St Croix CM, Dar HH, Mao G, Tyurin VA, Anthonymuthu TS et al (2021) PEBP1 wardens ferroptosis by enabling lipoxygenase generation of lipid death signals
Therapeutics 187 , 150166 (2018)