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The pathogenesis of glomerulonephritis involves a multitude of pathways including immune dysregulation, nephritogenic autoimmune responses, antigen complementarity, molecular mimicry, epitope spreading, and conformational changes [36]

Regulation of glutamate, GABA and dopamine transporter uptake, surface mobility and expression

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Semaglutide is a GLP-1 receptor agonist, which means it replicates the function of GLP-1, a naturally occurring hormone in the body

3,47 On electron microscopy, membrane-bound, electron-dense lysosomal granules are observed within hepatocytes, especially near the biliary pole, corresponding to the pigment seen on light microscopy