While Bcl-2, an apoptosis inhibitor, is deactivated by the translocation of Bax, an apoptosis promoter, to the mitochondrial membrane, certain ion channels are formed in the membranes
[1] These effects result from the medication's action on gastric motility and may be exacerbated by the appetite-suppressing effects of phentermine, potentially leading to inadequate nutritional intake, dehydration, or electrolyte imbalances
Jody Dushay is an assistant professor at Harvard Medical School and a physician at Beth Israel Deaconess Medical Center
J Am Chem Soc 134:60066017 Finklea HO (2001) Theory of coupled electron-proton transfer with potential-dependent transfer coefficients for redox couples attached to electrodes
Immunostaining After organotypic culture or cryosection, the cochlear explants or tissue sections were fixed with 4% PFA, permeabilized with 1% TritonX-100 in PBS, and blocked by incubation in PBT-1 solution (0.1% Triton X-100, 8% donkey serum, 1% bovine serum albumin, and 0.02% sodium azide in PBS) at room temperature for 1 h