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Moreover, these CD11c+ macrophages release pro-inflammatory markers such as TNF-a, IL-6, C-reactive protein (CRP), and monocyte chemotactic protein-1 (MCP-1), all of which exacerbate inflammation (55

After secretion, PYY 136 is metabolized by DPP-4 to form PYY 336 [75]

The triple receptor mechanism involving GLP-1, GIP, and glucagon activation creates a powerful synergy for glucose regulation that surpasses single or dual receptor approaches