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Attenuating astrocyte activation accelerates plaque pathogenesis in APP/PS1 mice

Wound healing and skin regeneration models

Similarly, for Si nano and bulk treatments, the TFC was higher in presence of 8 mg L -1 SiO 2 NPs compared to 4 mg L -1 Si bulk i.e

225 Administration of the PRMT4 inhibitor TP-064 in a mouse model of atherosclerotic cardiovascular disease can induce a decrease in monocyte tumor necrosis factor- (TNF-) secretion, downregulate the gene expression of the glycogen metabolism-related protein G6pc in the liver, and reduce plasma triglyceride levels, exerting regulatory effects from inflammatory and metabolic pathways

10.1111/j.1364-3703.2009.00558.x 181 ZhaoJ.WangJ.AnL.DoergeR