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The consequent ROS accumulation in the body is inevitably associated with tissue injury [1], since ROS are known to damage cell components, particularly DNA, leading to loss of function, induction of apoptosis, or even carcinogenesis [2]

By contrast, in the post-prandial phase, hepatic glucose production is reduced and hepatic glucose uptake is increased (enhanced glycogen-synthesis and the novo lipogenesis) because circulating levels of glucose are sufficient for the energy needs of non-hepatic organs (23, 24)

Its that shaking motion, not just the impact, that leads to the injury

Temporal profile of matrix metalloproteinases and their inhibitors after spontaneous intracerebral hemorrhage: relationship to clinical and radiological outcome

2015: Pickart et al