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Thats where malassezin comes in

One potential mechanism is the Nrf2-ARE pathway, which induces a phase 2 detoxification response that promotes disruption of Nrf2-Keap protein interactions, translocation of Nrf2 to the nucleus, and regulation of target gene expression through the ARE, enhancing cellular defense against oxidative damage (Sharma et al., 2010)

Though there is some human data, it's doesn't have the best "single-ingredient" evidence

Conflict of interest Authors BH, FW, and LQ were employed by Yunnan Botanee Bio-Technology Group Co., Ltd

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