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This site-specific modification is the key to SAGs ability to retain full biological activity
Philp D, Kleinman HK
10.1096/fj.02-0533fje FASEB J
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One very recent study of a TREM2 agonistic antibody in the cuprizone model reported enhanced myelin debris uptake and degradation, together with improved remyelination (Cignarella et al., Another approach could involve targeting lipid metabolism, as the uptake of too much cholesterol-rich myelin debris converts microglia into cells with a proinflammatory profile
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