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The IL-33 model was specifically selected to isolate ILC2-intrinsic responses independent of upstream epithelial-derived alarmin complexity present in the previously employed Alternaria model ( Figure 1 )

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It also modulates the activity of metalloproteinases (enzymes that break down extracellular matrix proteins) and their inhibitors, creating a balanced environment where damaged tissue is cleared while new tissue is being built

pH levels play a critical role in this process, and this is where many mixing mistakes occur