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This exosome-based signaling cascade transforms ferroptosis from a localized neuronal death event into a spatially expanding pathological phenomenon within neural networks

Pre-treatment with HTyr acetate, which is present in olive oil at a concentration similar to that of HTyr, significantly prevented oxidative stress-induced OPA1 cleavage and mitochondrial morphology changes, and this was accompanied by improvement of mitochondrial oxygen consumption capacity, ATP productive potential and activities of mitochondrial complexes I, II, and V, and inhibition of oxidative stress-induced MyHC decrease [314]

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