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Tumor necrosis factor (TNF) synthesized by palmitic acid-induced Kupffer cells, a fatty acid cargo of tumor-derived EVP, is translocated into hepatocytes and inhibits their fatty acid metabolism and OXPHOS [40]

Directly supplementing with oral reduced glutathione (L-GSH), however, presents a fundamental pharmacokinetic challenge: GSH has a plasma half-life of approximately 1.6 minutes, undergoes rapid hydrolysis in the gut by intestinal -glutamyl transpeptidase (-GT), and cannot cross cell membranes intact requiring full breakdown into constituent amino acids and intracellular resynthesis

They found no differences in p53 expression between irradiated cells treated with IF and controls, suggesting that IF likely acts through a p53-independent mechanism

doi: 10.3390/medsci10030038

Similar to CAF (Canas et al