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It has been recently shown [60] that T cells from depressed patients displayed a compromised metabolic profile accompanied by heightened gene expression of CTP1a (carnitine palmitoyltransferase 1 A), the mitochondrial enzyme responsible for ACs synthesis

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45,46,47 These comorbidities also pose challenges to disease modelling of ASD, as they may complicate the evaluation of ASD core behaviours in animal models

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doi: 10.1021/jf980985s