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Acknowledgements The authors appreciate and thank the efforts of the Center for the Development of Clinical Researches of the Educational and Therapeutic Research Complex of Birjand University of medical science

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In individuals exhibiting robust therapeutic outcomes, these microbial metabolites potentially initiate a complementary signaling cascade via free fatty acid receptors on enteroendocrine L-cells, thereby reinforcing host incretin pathways

How can Glutathione be administered

Knockdown of CPT1A can promote tumor cell susceptibility to Cisplatin