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Upregulated DRP1 in kidney diseases can promote the permeability of OMM via the interaction with Bcl-2-associated X protein (BAX), a crucial executor of mitochondria-regulated apoptosis (Spitz and Gavathiotis, 2022) to trigger cell apoptosis (Qin et al., 2019)

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It has become a major focus of biomedical research due to its potential roles in wound healing, angiogenesis, and anti-inflammatory signaling

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