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10.1056/NEJMoa2307563 68

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This formula is especially important for patients with GSTP1, GPX, and GSTM1 mutations

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While every effort has been made to ensure the accuracy of the information presented at the time of publication, Open Medscience does not guarantee the completeness or reliability of the content and accepts no responsibility for any consequences arising from its use

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If any symptom is sudden, severe, or persistent, contact your healthcare team promptly

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More recently, experimental evidence points oxidative stress and mitochondrial dysfunction as one of the common pathophysiological mechanisms responsible of neurotoxicity in CIPN

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