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doi: 10.1016/j.msec.2018.12.135 104 RouhiS

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Our data provide important new information concerning potential interactions between GSSG, protein glutathionylation, and TNF-induced hepatotoxicity, as well as its critical involvement in the pathogenesis of NAFLD

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Pickart originally isolated GHK as the "growth factor" responsible for restoring proliferation to liver cells in older-donor plasma, and the copper-binding role was recognized shortly afterward as the molecular basis for many of the activities attributed to the peptide

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